Ryan O'Hare Doig

Ryan O'Hare Doig
Adelaide Medical School
Faculty of Health and Medical Sciences

Ryan’s early research career has focused on understanding the pathophysiology of secondary degeneration following neurotrauma to the central nervous system. Ryan looks to help develop new techniques to provide a more accurate diagnosis and prognosis of spinal cord injury, to identify potential treatment strategies in a clinical setting.

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Ryan O'Hare Doig

Ryan’s early research career has focused on understanding the pathophysiology of secondary degeneration following neurotrauma to the central nervous system. Ryan looks to help develop new techniques to provide a more accurate diagnosis and prognosis of spinal cord injury, to identify potential treatment strategies in a clinical setting.

Appointments

Date Position Institution name
2017 Adjunct Lecturer University of Adelaide, Adelaide
2017 Post-doctoral Research Officer South Australian Health and Medical Research Institute, Adelaide

Education

Date Institution name Country Title
2017 University of Western Australia, Perth Australia PhD Neuroscience & Physiology
2012 University of Western Australia, Perth Australia BSc (Neuroscience) with First Class Honours

Research Interests

Neuroscience, Behaviour and Brain Health, Animal Cell and Molecular Biology, Animal Neurobiology, Brain injury, Central Nervous System, Neurosciences, Nuclear Medicine & Medical Imaging, Spinal cord, Spine

Journals

Year Citation
2017 O'Hare Doig, R. L., Chiha, W., Giacci, M. K., Yates, N. J., Bartlett, C. A., Smith, N. M. ... Fitzgerald, M. (2017). Specific ion channels contribute to key elements of pathology during secondary degeneration following neurotrauma.. BMC neuroscience, 18, 1, 62-.
10.1186/s12868-017-0380-1
2017 Yates, N. J., Giacci, M. K., O'Hare Doig, R. L., Chiha, W., Ashworth, B. E., Kenna, J. ... Fitzgerald, M. (2017). Delayed treatment of secondary degeneration following acute optic nerve transection using a combination of ion channel inhibitors.. Neural regeneration research, 12, 2, 307-316.
10.4103/1673-5374.200814
2016 O'Hare Doig, R. L., Bartlett, C. A., Smith, N. M., Hodgetts, S. I., Dunlop, S. A., Hool, L. & Fitzgerald, M. (2016). Specific combinations of ion channel inhibitors reduce excessive Ca(2+) influx as a consequence of oxidative stress and increase neuronal and glial cell viability in vitro.. Neuroscience, 339, 450-462.
10.1016/j.neuroscience.2016.10.005
2015 Meloni, B. P., Milani, D., Edwards, A. B., Anderton, R. S., O'Hare Doig, R. L., Fitzgerald, M. ... Knuckey, N. W. (2015). Neuroprotective peptides fused to arginine-rich cell penetrating peptides: Neuroprotective mechanism likely mediated by peptide endocytic properties.. Pharmacology & therapeutics, 153, 36-54.
10.1016/j.pharmthera.2015.06.002
2015 Challenor, M., O'Hare Doig, R., Fuller, P., Giacci, M., Bartlett, C., Wale, C. H. ... Fitzgerald, M. (2015). Prolonged glutamate excitotoxicity increases GluR1 immunoreactivity but decreases mRNA of GluR1 and associated regulatory proteins in dissociated rat retinae in vitro. Biochimie, 112, 160-171.
10.1016/j.biochi.2015.03.008
2015 O'Hare Doig, R. L. & Fitzgerald, M. (2015). Novel combinations of ion channel inhibitors for treatment of neurotrauma.. Discovery medicine, 19, 102, 41-47.
2014 O'Hare Doig, R. L., Bartlett, C. A., Maghzal, G. J., Lam, M., Archer, M., Stocker, R. & Fitzgerald, M. (2014). Reactive species and oxidative stress in optic nerve vulnerable to secondary degeneration.. Experimental neurology, 261, 136-146.
10.1016/j.expneurol.2014.06.007
2013 Szymanski, C. R., Chiha, W., Morellini, N., Cummins, N., Bartlett, C. A., O'Hare Doig, R. L. ... Fitzgerald, M. (2013). Paranode Abnormalities and Oxidative Stress in Optic Nerve Vulnerable to Secondary Degeneration: Modulation by 670 nm Light Treatment.. B. V. Bui (Ed.). PloS one, 8, 6, e66448-.
10.1371/journal.pone.0066448
2013 Savigni, D. L., O'Hare Doig, R. L., Szymanski, C. R., Bartlett, C. A., Lozić, I., Smith, N. M. & Fitzgerald, M. (2013). Three Ca2+ channel inhibitors in combination limit chronic secondary degeneration following neurotrauma.. Neuropharmacology, 75, 380-390.
10.1016/j.neuropharm.2013.07.034

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