Natasha Prizzi

Dr Natasha Prizzi

Research Associate

Centre for Cancer Biology

College of Health

Eligible to supervise Masters and PhD (as Co-Supervisor) - email supervisor to discuss availability.

I am currently a post-doctoral research associate in the Tumour Microenvironment Laboratory at Adelaide University in South Australia, with research expertise in cutaneous biology. I specialise in the cellular and matrix interactions that can modify the cutaneous microenvironment, resulting in changes in susceptibility to tumour initiation and efficacy in wound healing. 
 

Dr Kolesnikoff is a  post-doctoral research associate in the Tumour Microenvironment Laboratory at the Adelaide University, with research expertise in cutaneous biology. She specialises in the cellular and matrix interactions that can modify the cutaneous microenvironment, resulting in changes in susceptibility to tumour initiation and efficacy in wound healing. 

Date Type Title Institution Name Country Amount
2024 Award 2024 AWTRS Outstanding ECR Presentation Australasian Wound and Tissue Repair Society Australia -
2024 Award 2024 MBSANZ Bob Fraser New Investigator Award Matrix Biology Society for Australia and New Zealand Australia -

Date Institution name Country Title
2013 University of Adelaide Australia PhD
2008 Flinders University Australia Bachelor of Biotechnology (Honours)

Year Citation
2024 Johan, M. Z., Pyne, N. T., Kolesnikoff, N., Poltavets, V., Esmaeili, Z., Woodcock, J. M., . . . Samuel, M. S. (2024). Accelerated Closure of Diabetic Wounds by Efficient Recruitment of Fibroblasts upon Inhibiting a 14-3-3/ROCK Regulatory Axis. The Journal of Investigative Dermatology, 144(11), 2562-2573.e4.
DOI Scopus1 WoS1 Europe PMC1
2022 Kolesnikoff, N., Chen, C. -H., & Samuel, M. S. (2022). Interrelationships between the extracellular matrix and the immune microenvironment that govern epithelial tumour progression. Clinical Science, 136(5), 361-377.
DOI Scopus38 WoS36 Europe PMC36
2020 Boyle, S. T., Poltavets, V., Kular, J., Pyne, N. T., Sandow, J. J., Lewis, A. C., . . . Samuel, M. S. (2020). ROCK-mediated selective activation of PERK signalling causes fibroblast reprogramming and tumour progression through a CRELD2-dependent mechanism. Nature Cell Biology, 22(7), 882-895.
DOI Scopus60 WoS60 Europe PMC57
2020 Boyle, S. T., Poltavets, V., Kular, J., Pyne, N. T., Sandow, J. J., Lewis, A. C., . . . Samuel, M. S. (2020). Publisher Correction: ROCK-mediated selective activation of PERK signalling causes fibroblast reprogramming and tumour progression through a CRELD2-dependent mechanism.. Nat Cell Biol, 22(7), 908.
DOI Scopus5 WoS4 Europe PMC6
2016 Yip, K., Kolesnikoff, N., Hauschild, N., Biggs, L., Lopez, A., Galli, S., . . . Grimbaldeston, M. (2016). The Nedd4-2/Ndfip1 axis is a negative regulator of IgE-mediated mast cell activation. Nature Communications, 7(article no. 13198), 13198-1-13198-14.
DOI Scopus32 WoS31 Europe PMC27
2015 Tong, P., Roediger, B., Kolesnikoff, N., Biro, M., Tay, S., Jain, R., . . . Weninger, W. (2015). The skin immune atlas: three-dimensional analysis of cutaneous leukocyte subsets by multiphoton microscopy. Journal of Investigative Dermatology, 135(1), 84-93.
DOI Scopus103 WoS92 Europe PMC84
2015 Kular, J., Scheer, K., Pyne, N., Allam, A., Pollard, A., Magenau, A., . . . Samuel, M. (2015). A negative regulatory mechanism involving 14-3-3ζ limits signaling downstream of ROCK to regulate tissue stiffness in epidermal homeostasis. Developmental Cell, 35(6), 759-774.
DOI Scopus31 WoS25 Europe PMC30
2014 Kolesnikoff, N., Attema, J., Roslan, S., Bert, A., Schwarz, Q., Gregory, P., & Goodall, G. (2014). Specificity protein 1 (Sp1) maintains basal epithelial expression of the miR-200 family: implications for epithelial-mesenchymal transition. Journal of Biological Chemistry, 289(16), 11194-11205.
DOI Scopus61 WoS57 Europe PMC53
2014 Yip, K., Kolesnikoff, N., Yu, C., Hauschild, N., Taing, H., Biggs, L., . . . Grimbaldeston, M. (2014). Mechanisms of vitamin D₃ metabolite repression of IgE-dependent mast cell activation. Journal of Allergy and Clinical Immunology, 133(5), 1356-1379.
DOI Scopus103 WoS89 Europe PMC78
2013 Attema, J., Bert, A., Lim, Y., Kolesnikoff, N., Lawrence, D., Pillman, K., . . . Goodall, G. (2013). Identification of an enhancer that increases miR-200b~200a~429 gene expression in breast cancer cells. PLoS One, 8(9), 1-15.
DOI Scopus21 WoS19 Europe PMC19
2008 Paterson, E., Kolesnikoff, N., Gregory, P., Bert, A., Khew-Goodall, Y., & Goodall, G. (2008). The microRNA-200 Family Regulates Epithelial to Mesenchymal Transition. The Scientific World Journal, 8, 901-904.
DOI Scopus76 WoS72 Europe PMC61
2008 Bracken, C., Gregory, P., Kolesnikoff, N., Bert, A., Wang, J., Shannon, M., & Goodall, G. (2008). A double-negative feedback loop between ZEB1-SIP1 and the microRNA-200 family regulates epithelial-mesenchymal transition. Cancer Research, 68(19), 7846-7854.
DOI Scopus963 WoS910 Europe PMC860
  • How can we predict bowel cancer recurrence?, Cancer Council SA - Beat Cancer, 01/01/2023 - 31/12/2023

Date Role Research Topic Program Degree Type Student Load Student Name
2025 Co-Supervisor Understanding how the epigenetic field effect exerted by cancers upon their microenvironment promotes tumour progression Doctor of Philosophy Doctorate Full Time Mrs Zahra Esmaeili

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